Anti-cancer chronic atrophic gastritis

November 25, 2009 · Posted in Prevention and care of chronic gastritis 

Chronic gastritis (superficial gastritis and atrophic gastritis) is a common diseases and frequently-occurring disease. Endoscopic survey confirmed that our population the incidence of chronic gastritis as high as 60% or more, atrophic gastritis which accounts for about 1 / 5.

Atrophic gastritis, gastric cancer in the past was considered to be a prelude to (precancerous lesions), and now see, this viewpoint is biased. However, atrophic gastritis and gastric cancer have definite relations, which is based on: ① studies have shown that, for atrophic gastritis endoscopic biopsies drawn that there was “colonic type intestinal metaplasia” and “atypical hyperplasia” It’s two kinds of gastric mucosal lesions , it may develop into gastric cancer. This is acknowledged. ② epidemiological investigation, in gastric cancer high places, the crowd, atrophic gastritis in a high incidence. ③ pathological examination found that the surrounding mucosa of gastric cancer, atrophic lesions more common.

It can be said, atrophic gastritis, though not pre-cancerous lesions, but if left to natural development, there are a few cases may develop into gastric cancer. Therefore, we must take measures to seriously deal with, so that condition remains stable (complete cure of this disease difficult), in order to avoid the occurrence of cancer.

Atrophic gastritis cancer prevention (for cancer, the treatment also is a preventive) measures are:

1. Antimicrobial therapy. Today the medical profession recognized as Helicobacter pylori (HP) is certainly a pathogen of chronic gastritis, it should first of antimicrobial therapy.

2. Oral mucosal protective agent. Commonly used drugs are: sucralfate, and allowing the formation of gastric mucosal mucin complex protective film to protect the gastric mucosa; gastric mucin can be formed in the stomach mucous membrane covered surface, reduce bile reflux on gastric mucosal stimulation; chlorophyll, can promote the role of gastric mucosal inflammation receded protection; monkey mushroom pieces can protect gastric mucosa.

3. To improve the concentration of acid. Atrophic gastritis, or lack of regular acid-free acid (gastric cancer showed acid-free state) can be used pepsin mixture or dilute hydrochloric acid mixture; five low-dose intramuscular injection of pentagastrin, there are nourishing, protecting gastric mucosa and to promote the role of parietal cells secrete hydrochloric acid .

4. Weimeisu clothes. Can improve human immunity, enhance the activity of the human body detoxification enzymes, inhibit cancer cell growth and prevent abnormal cell metabolism.

5. Treatment of bile reflux. In the pyloric sphincter dysfunction, or when the stomach ~ jejunum anastomosis may be due to the long-term destruction of bile reflux and gastric mucosal barrier, resulting in chronic superficial gastritis, and then develop into a chronic atrophic gastritis. In this case, can be applied to gastric motility drug, to prevent bile reflux, so as to achieve the purpose of protection of gastric mucosa.

6. Diet. Low stomach acid and bile reflux were advised to eat lean meat, poultry, fish, milk and other high-protein low-fat diet; to be slowly, bogey eating and drinking; avoid long-term drinking tea, alcohol ( in particular, alcohol), coffee and eat spicy, hot and rough food.

7. The elimination of certain pathogenic incentives. Such as smoking cessation, avoiding long-term use of the gastric mucosa are stimulated by drugs (such as sodium salicylate, indomethacin, phenylbutazone and aspirin, etc.), ease mental stress, maintain a positive mood, thereby enhancing immune function and increased disease resistance .

8. On a regular basis review. Pairs of atrophic gastritis with incomplete colonic type intestinal metaplasia and dysplasia in patients with gastroscopy should be done on a regular basis for review: general atrophic gastritis, a second three-year review, incomplete colonic type intestinal metaplasia with mild dysplasia in 1 1 times, with moderate dysplasia in 3 months, 1 times, with severe atypical hyperplasia (cancer rate of 10% or more) should be regarded as cancerous, may be surgical excision.

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